I dont think researchers consider alexithymia an ASD (do they?). Nevertheless its looking likely that alexithymia is a core symptom on all the diagnostic criteria for AS, and the forthcoming DSM-V may spell this out explicitly.
Current diagnostic wordings referring to alexithymia-like symptoms:
DSM-IV: Lack of social or emotional reciprocity
ICD-10: Impaired or deviant response to other people's emotions
Szatmari: Difficulty sensing feelings of others
NAS definition: Difficulties expressing themselves, especially when talking about emotions
Gillberg criteria: Emotionally inappropriate behavior. Impairment of comprehension.
Another development is that the DSM-V research committee recently published a paper online saying they recommend "deleting Asperger's Disorder" from the current list of disorders because it is not sufficiently different from HFA, and instead just employ High Functioning Autism label for AS. If they do this, then those of us with the AS diagnosis will no longer be AS !
Quote:
Autism and Other Pervasive Developmental Disorders Conference (February 3-5, 2008)
Prepared by Michael B. First, M.D., DSM Consultant to the American Psychiatric Institute for Research and Education (APIRE), a subsidiary of the American Psychiatric Association
The fifth panel addressed the question Asperger’s Disorder – is it Autism? In her introduction, Francesca G. Happé, Ph.D., (London, UK) raised some of the key questions that have arisen regarding the diagnosis of Asperger’s Disorder, which was introduced into DSM-IV in 1994. These questions include: is there an ‘Asperger’ subgroup of autism with distinct cause, course, cognitive profile, and intervention needs, and if so, what is its relation to other ASDs? Asperger’s disorder is essentially defined as meeting criteria for autism without the language impairment. Lorna Wing introduced the term in 1981 to aid recognition of the part of the autism spectrum with good IQ and language. It has increased awareness and recognition and helped to clarify the core deficits of ASD, but also increases the possibility that there may be over-diagnosis of ASD. Asperger’s disorder has also had an impact on family studies of autism with regard to what we recognize as “caseness.” Dr. Happe noted that the current criteria do not work: they do not allow for developmental change, the early language criteria do not demarcate groups with different prognoses, it is hard to apply the diagnosis for adult cases, and there is no clear conceptual basis for the diagnosis. Dr. Happe concluded that although there is a recognizable Asperger’s type and that some cases of classic autism grow into this picture, she wonders whether there may be a better classification schema. Sally Ozonoff, Ph.D., (Sacramento, CA), in her presentation, compared high functioning autism (HFA) with Asperger’s, and noted that there were few differences in their definitional DSM-IV criteria; both require two social symptoms and one repetitive/stereotyped symptom, both are in the average range intellectually and have current fluent language. The main criterion distinguishing the two disorders is the requirement in Asperger’s that onset of language occurs at the expected time, e.g., single words by age 2. Dr. Ozonoff noted that it is difficult to evaluate the literature since definitions vary across studies and that many children who are thought clinically to have Asperger’s actually meet criteria for autism (which supercedes a diagnosis of Asperger's). There is some evidence to suggest that Asperger's and HFA do not represent distinct disorders: they co-occur in the same families and do not “breed true” (i.e., family members of patients with Asperger's have HFA and family members of patients with HFA have Asperger's); children with autism who develop language have similar outcome to Asperger's; HFA and Asperger's are indistinguishable by school-age; and although studies find better language skills and/or verbal IQ in Asperger's, multiple studies have found no group differences in other neuropsychological domains.
The third breakout group made the following recommendations: 1) delete Asperger’s disorder; 2) delete CDD; and 3) create an ASD with two types: Type I would be for prototypical cases characterized by problems in social interaction, social communication, and repetitive behaviors or preoccupations, and Type II is for atypical cases.
On a completely different subject, according to Tania Singer the proverbial lack of empathy in AS is ONLY in those who have alexithymia:
Asperger’s syndrome may not lead to lack of empathy
By Tina Hesman Saey
Web edition : Thursday, April 24th, 2008
People with autism can feel others’ pain, two new studies show.
Autism robs people of social and language skills and locks people in their own worlds. Scientists thought that the social defects were due in part to an autistic person’s inability to determine what other people think and feel and adjust behavior accordingly. But new research from scientists in Switzerland and the Netherlands shows that the brains of people with Asperger’s syndrome or other high-functioning autism spectrum disorders do respond with empathy to others’ pain or emotion.
Tania Singer of the University of Zurich in Switzerland and her colleagues studied people who have trouble identifying their own emotions, a condition known as alexithymia. “You need to understand your own feelings to understand the feeling of other people,” Singer says.
The disorder isn’t well known and often doesn’t interfere with people’s lives, Singer said April 14 in San Francisco at a meeting of the Cognitive Neuroscience Society. About 10 percent of people in the general population may have alexithymia. But people with Asperger's syndrome are far more likely to have alexithymia, with 60 to 80 percent of this group having the condition.
The researchers first tested couples to find out how empathy works in the brain normally. One partner was in a brain scanner known as an functional MRI, which uses magnetic fields to measure blood flow in the brain, an indicator of brain activity. Researchers made the person feel pain in a hand. A part of the brain called the insula, involved in linking body responses and emotion, lit up in response to the pain. When the partner of the person in the scanner experienced pain, the insula of the partner in the scanner lit up in sympathy.
But in people who have alexithymia, their insulas didn’t activate when they saw their partners in pain. The insulas of people with Asperger's syndrome also lit up when they saw others in pain, but only when they were in touch with their own emotions.
The result may mean that researchers and clinicians should rethink the deficit in empathy associated with Asperger’s syndrome. “I would just claim not all Asperger's [patients] will have an empathy deficit,” Singer says. It’s the degree to which people have alexithymia that determines whether they have an empathy deficit.
Just when I felt sure I knew the subject!